DAngel1111 @Angel1111D

@Gavtaraysin Greatest update Broden!

“🚨NORWEGIAN CONFIRMATION BOMBSHELL: COVID infection leaves lasting, hidden scars on the heart muscle, even years later.” A Cardiovascular & Thoracic Surgeon is trying to warn you. But you keep listening to politicians & billionaires.

Harry Spoelstra @HarrySpoelstra

4 weeks ago

Impact of SARS-CoV-2 infection on subclinical myocardial injury in the general population: the Trøndelag Health Study 🚨NORWEGIAN CONFIRMATION BOMBSHELL: COVID infection leaves lasting, hidden scars on the heart muscle, even years later. "An elevated cardiac troponin I (cTnI)

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📉 Wastewater numbers are currently incredibly low, about *7 times* lower than the spring lulls in years past. We're approaching flu-in-the-summer style prevalence.

Search interest for brain fog on Google recently hit an all time high.

Long COVID Far More Common Than Records Show, and It's Getting Worse share.google/KBz4ohtOLz3qEU…

@WyattCatarina I’ve never seen her having the same meal twice!

Tissue-specific autoantibody signatures reveal immune alterations undetected by routine serology in long COVID 🚨83% of long COVID patients have rogue autoantibodies attacking their own heart, lungs & blood vessels, and every standard blood test misses it completely. VERY INTERESTING! ➡️In a UNIQUE Hungarian cohort of 114 long COVID patients versus 36 pre-pandemic controls, tissue-specific Western blotting detected autoantibodies in 83% of cases, with strong cardiovascular dominance, ➡️Vascular autoreactivity was markedly higher in long COVID (34% vs. 8%, p<0.05), cardiac (54%) and pulmonary (34%) signals trended elevated but did not reach significance( cohort size?), ➡️Autoantibodies were predominantly IgM-skewed, polyreactive (up to 8 bands per patient), and persisted longitudinally (mean 141 days), with new isotypes emerging over time, ➡️Standard ANA testing showed no group differences and zero clinical correlations, rendering it useless for detecting these alterations, ➡️Cardiac autoreactivity associated with hypertension and headache, overall autoreactivity correlated with anosmia/ageusia, female sex, CRP, BMI, creatinine, and troponin levels, ➡️The study used human cardiac, pulmonary, and vascular tissue homogenates. ➡️Findings were independent of routine serology and highlight an under-recognized immune component invisible to current diagnostics. ➡️“This persistent, IgM-skewed profile suggests ongoing immune dysregulation and may reflect a previously underrecognized component of the immunological response in long COVID, highlighting the need for targeted immunodiagnostic approaches beyond routine serology.” ‼️Why this is shocking: It proves that in 83% of long COVID patients, the immune system is actively producing autoantibodies that directly target their own heart, lung, and especially blood-vessel tissues, yet every standard blood test (ANA HEp-2) comes back normal. These rogue antibodies are polyreactive, IgM-dominant, persist for months, and keep evolving. They correlate with real symptoms (anosmia, hypertension, headache) and lab markers of damage (troponin, CRP). ‼️In other words: The majority of long COVID sufferers have smouldering, organ-specific autoimmunity that is completely invisible to routine diagnostics. Doctors are flying blind while patients’ tissues are quietly under autoimmune attack. 🤔As far as I know, this is the first direct evidence of hidden, cardiovascular-dominant tissue autoimmunity driving the chronic L0ngC0vid phase! #BookMark #AvoidSars2 #AvoidReinfections link.springer.com/article/10.100…

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@nikitabier @BillyM2k I am tired of screening and blocking fake accounts-but never regret masking.

BREAKING: A former senior advisor to Anthony Fauci has been INDICTED for his role in the COVID-19 coverup This guy might throw Anthony Fauci under the BUS 👀 David Morens and his co-conspirators FALSIFIED records in an effort to SUPPRESS the lab-leak theory, and used his personal Gmail account rather than his NIH email in order to avoid being FOIA'd Fauci might just be next!

57% of people with long COVID reported new eye issues Blurred vision, dry eyes, floaters or flashes

Denis - The COVID info guy - @BigBadDenis

3 months ago

"Eye symptoms may signal higher-severity long COVID" Yale researchers found 57% of people with long COVID reported new eye issues—like blurred vision, dry eyes, floaters or flashes—after their initial infection. cidrap.umn.edu/covid-19/eye-s…

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@OrangeCatsRule1 I went to a community event and was shushed twice-they told me I was talking too loud bc mask. Lmao

@serahaldin @bitterbichon This hits home.

@LatidosDePasion Ah, Im sure it will come in great after the bunnies and squirrels 🐿️ feast.

Just got signed back in after new phone…what did I miss?!? 🥸

🚨⚠️ Heart attacks🫀in people under 40 have risen 66% since the beginning of the COVID pandemic. COVID is a vascular disease COVID can affect the cardiovascular system by increasing blood stickiness, which raises the likelihood of blood clot formation.

@DarainChains Sending love Dara 0000x

@danka_mm @Sreneaurn73 @HarrySpoelstra Almost every doctor recommends-its infuriating

@Sreneaurn73 @HarrySpoelstra So true!

Post-exertional malaise and the myth of cardiac deconditioning: rethinking the pathophysiology of long covid ‼️Important and HIGH TIME this got addressed! ➡️This excellent international paper scientifically argues that heart-related issues seen in long COVID (like problems with blood flow/preload, inflammation, scarring, or poor oxygen use by muscles) aren't mainly explained by simple deconditioning from bed rest or inactivity. Instead, deeper problems are likely involved, such as issues with mitochondria (the cell's energy factories), blood vessel/endothelial dysfunction, autoimmunity, or other post-viral effects. ➡️The IMPORTANT takeaways for patients and colleagues: 1. Long COVID is NOT just "being out of shape" or simple deconditioning from illness/inactivity. The idea that heart and exercise problems in long COVID are mostly reversible deconditioning is a myth, it fails to explain what doctors actually see. 2. Post-exertional malaise (PEM) is the REAL hallmark, hitting ~80% of long COVID patients hard. Even mild activity (physical or mental) triggers a delayed, brutal crash: massive fatigue, pain, brain fog, etc., that drags on for days, weeks, or longer, sometimes permanently worsening your baseline. This is not what happens in normal deconditioning. 3. Heart issues in long COVID go way beyond deconditioning. We're talking real damage: myocardial scarring, inflammation, "leaky" blood vessels, preload failure (heart doesn't fill properly), mitochondrial dysfunction, endothelial problems, autoimmunity, and poor oxygen use in muscles. Deconditioning alone cannot account for these, especially the weird preload issues seen on invasive testing. 4.Pushing graded exercise therapy is DANGEROUS for people with PEM. Traditional "build up gradually" programs are contraindicated — they risk serious harm, crashes, and permanent setbacks. The WHO already warns against this for anyone with PEM. 5. One-size-fits-all exercise advice is reckless. - Stop assuming long COVID = deconditioning. - Screen EVERY patient for PEM first (using tools like questionnaires). - Treatment must be highly personalized: pacing (staying strictly within your energy limits, often with heart rate monitoring), light tolerated activity, breathing work, avoiding prolonged bed rest, and sometimes meds for related issues like POTS. ‼️Long COVID's exercise intolerance and cardiac changes are largely independent of deconditioning. Ignoring PEM and pushing standard rehab can actively harm patients. Doctors and patients need to ditch the outdated "just get fitter" mindset and adopt physiology-guided, PEM-safe strategies, or risk making things much worse. bjsm.bmj.com/content/early/…